Procurou por: Cefsulodin+sodium+salt

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1-Amino-4-bromoanthraquinone-2-sulphonic acid sodium salt ≥98.0% (por HPLC)

Fornecedor: TCI

Descrição: 1-Amino-4-bromoanthraquinone-2-sulphonic acid sodium salt ≥98.0% (by HPLC)

Cytidine-5'-diphosphocholine sodium salt (CDP choline) ≥98.0% (por HPLC)

Fornecedor: TCI

Descrição: Cytidine-5'-diphosphocholine sodium salt (CDP choline) ≥98.0% (by HPLC)

1-Butanesulphonic acid sodium salt 99+% para HPLC

Fornecedor: Thermo Fisher Scientific

Descrição: 1-Butanesulphonic acid sodium salt 99+% para HPLC

MSDS

Primuline sodium salt, pó C.I. 49000

Fornecedor: MP Biomedicals

Descrição: Moderately soluble in water. Soluble in methanol(1 mg/ml): yellow to yellow-green solution.

Carboxymethyl cellulose sodium salt FCC, baixa viscosidade

Código de Artigo: (SPCMC1103-2.5KG)

Fornecedor: Spectrum Chemical

Descrição: Carboxymethyl cellulose sodium salt FCC, baixa viscosidade

UOM: 1 * 2,5 kg

Promoção

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Cefotaxime sodium salt ≥95%, pó quase branco

Fornecedor: MP Biomedicals

Descrição: Cefotaxime is a cephalosporin, which acts by interference of synthesis of peptidoglycan of the bacterial cell wall. Cefotaxime, a cephalosporin antibiotic, is active against both gram-negative and gram-positive bacteria. In addition, cefotaxime blocks the division of cyanobacteria and lower plant organelles/plastids such as the photosynthetic organelles of Glaucophytes and chloroplasts of bryophytes.
Cefotaxime is used for infections of the respiratory tract, skin, bones, joints, urogenital system, meninges, and bloodstream. It generally has good coverage against most Gram-negative bacteria, with the notable exception of Pseudomonas. It is also effective against most Gram-positive cocci except for Enterococcus. It is active against penicillin-resistant strains of Streptococcus pneumoniae. It has modest activity against the anaerobic Bacteroides fragilis. In meningitis, cefotaxime crosses the blood–brain barrier better than cefuroxime.
Cefotaxim inhibits bacterial cell wall synthesis by binding to penicillin-binding proteins (PBPs) which inhibits the final transpeptidation step of peptidoglycan synthesis in bacterial cell walls. As a result, bacteria lyse due to cell wall autolytic enzymes.

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